Category: Transient Receptor Potential Channels

No major differences were detected in p38-deficient muscle at the same neonatal stage, in spite of the reduced fusion index observed (2005); p38 f/f, p38 f/f, p38 f/f will be explained elsewhere. increased cyclin D1 transcription, whereas inhibition of JNK activity rescued the proliferation phenotype. Thus, p38 controls myogenesis by antagonizing the activation of the […]